911 THE HYPERTROPHIC SEPTAL PERFORATING BRANCH MISINTERPRETED AS INTERVENTRICULAR DEFECT

Abstract Introduction the septal perforating arteries of heart usually branch off from anterior and inferior interventricular supply septum conduction system therein. Aim we present case hypertrophic in a 71-year-old man who experienced dyspnoea which was initially misinterpreted as defect. Materials methods transthoracic echocardiography without with contrast, coronary angiography angioTC 3D reconstructions were performed. Results showed dilative cardiopathy EF 30%, akinesia IVS, apical wall. Hypokinesia remaining walls. At color-Doppler: severe mitral regurgitation moderate tricuspidal regurgitation. Coronary multivessel atherosclerotic artery disease. echocardiographic control reported presence turbulent flows at level medio-basal segment posterior IVS. Echo-power-Doppler no shunts. For persisting suspect defect performed contrast that hypoperfusion IVS after infusion but clear signs To complete diagnostic iter finally CT-Scan long intramyocardial course septum, detected Conclusions Septal have large variability their anatomy. Particular clincal conditions such chronic syndromes could lead to reactive hypertrophy. Is important recognise it due its similarity or myocardial bridging effect. A better understanding these will help physicians enhance cardiac care for patients.